Transfidelity Price (FIDEL)

The live Transfidelity (FIDEL) price today is $ 0.203374, with a 4.15% change over the past 24 hours. The current FIDEL to USD conversion rate is $ 0.203374 per FIDEL.
Transfidelity currently ranks #- by market capitalization at $ 203,374, with a circulating supply of 1.00M FIDEL. During the last 24 hours, FIDEL traded between $ 0.203433 (low) and $ 0.214101 (high), reflecting market activity. Its all-time high stands at $ 0.8633, while the all-time low was $ 0.194966.
In short-term performance, FIDEL moved -0.25% in the last hour and -18.89% over the past 7 days. Over the past day, total trading volume reached --.
The current Market Cap of Transfidelity is $ 203.37K, with a 24-hour trading volume of --. The circulating supply of FIDEL is 1.00M, with a total supply of 1000000.0. Its Fully Diluted Valuation (FDV) is $ 203.37K.
-0.25%
-4.15%
-18.89%
-18.89%
During today, the price change of Transfidelity to USD was $ -0.008809312060989.
In the past 30 days, the price change of Transfidelity to USD was $ -0.0807407795.
In the past 60 days, the price change of Transfidelity to USD was $ -0.1392030967.
In the past 90 days, the price change of Transfidelity to USD was $ -0.514994749339855.
| Period | Change (USD) | Change (%) |
|---|---|---|
| Today | $ -0.008809312060989 | -4.15% |
| 30 Days | $ -0.0807407795 | -39.70% |
| 60 Days | $ -0.1392030967 | -68.44% |
| 90 Days | $ -0.514994749339855 | -71.68% |
In 2040, the price of Transfidelity could potentially see a growth of 0.00%. It could reach a trading price of $ --.
Neurodegenerative diseases such as Alzheimer's Disease are devastating conditions that impact millions globally. These diseases are driven by the misfolding and aggregation of proteins, which lead to brain cell death and cognitive decline. To make matters worse, as we age our cells become more error-prone, resulting in more errors during protein printing, causing proteins to clump together and form aggregates.
Project Transfidelity aims to solve this by improving the accuracy of protein printing, preventing toxic aggregates from forming. Led by Dr. Dimitri Scherbakov and Dr. Rashid Akbergenov, with over 50 years of combined experience in the lab, we have identified two molecules that may improve the efficiency of protein production, potentially preventing protein aggregates, hypothesized to cause dementia.
Despite the significant research funding allocated to neurodegenerative diseases, current therapies continue to treat the symptoms caused by the protein aggregates, with little progress made to stop or reverse disease. We’re trying to go after a potential root cause - rather than trying to clear aggregates once they are there, we aim to prevent them from forming in the first place by reducing the production of faulty proteins. With a clear path towards developing novel intellectual property (IP) and an ever-growing target market, we believe we are poised to make an impact on neurodegenerative disorders and the way we age.
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